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m6A-mediated upregulation of miRNA-193a aggravates cardiomyocyte apoptosis and inflammatory response in sepsis-induced cardiomyopathy via the METTL3/ miRNA-193a/BCL2L2 pathway

InflammationCardiomyopathyMolecular PhysiologyMirna-193a AggravatesApoptosisImmunologyInflammatory ResponseCell DeathMettl3/ Mirna-193a/bcl2l2 PathwaySmall RnaMicrorna DetectionMedicineCell BiologyCell Signaling