<scp>TL1A</scp> promotes the postoperative cognitive dysfunction in mice through <scp>NLRP3</scp>‐mediated <scp>A1</scp> differentiation of astrocytes

Abstract

Our results demonstrate that TL1A plays an important role in POCD in mice, which promotes the A1 differentiation of astrocytes through NLRP3, thereby exacerbating the progression of cognitive dysfunction.