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Notoginsenoside R1 intervenes degradation and redistribution of tight junctions to ameliorate blood-brain barrier permeability by Caveolin-1/MMP2/9 pathway after acute ischemic stroke

Cerebral Vascular RegulationIschemic StrokeBlood–brain BarrierMedicinePharmacologyBlood-brain Barrier PermeabilityVascular BiologyNeuroprotectionNeurologyNeuroscienceNotoginsenoside R1Cerebral Blood FlowTight JunctionsStrokeIschemic SyndromeReperfusion InjuryNeuroinflammation