Elimination of endogenous high molecular weight FGF2 prevents pressure-overload-induced systolic dysfunction, linked to increased FGFR1 activity and NR1D1 expression - Concepedia

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Elimination of endogenous high molecular weight FGF2 prevents pressure-overload-induced systolic dysfunction, linked to increased FGFR1 activity and NR1D1 expression

10

Citations

44

References

2021

Year

Navid Koleini, Barbara E. Nickel, Raghu S. Nagalingam, Natalie M. Landry, Robert R. Fandrich, David Cheung, Ian Dixon, Michael P. Czubryt, Davinder S. Jassal, Peter A. Cattini,

Cell and Tissue Research

References

	Year	Citations

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