Natalie M. Landry - Concepedia

Author

Natalie M. Landry

University of Manitoba ORCID

Also Known As

Landry, Natalie, Natalie Landry, Natalie M Landry, Natalie M. Landry

Publications

462

Citations

H-Index

Concepts

All Affiliations

University of Manitoba St. Boniface Hospital Children's Hospital Research Institute of Manitoba

About

Natalie M. Landry is an author at University of Manitoba specializing in fibrosis, cardiology, and medicine.

Top Concepts

#	Concept	H-Index	Publications	Citations
1	Medicine	2	2	34
2	Cell Biology	2	2	34
3	Biomedical Engineering	1	1	14
4	Cellular Physiology	1	1	14
5	Cardiac Regeneration	1	1	14

Natalie M. Landry

Publications

267

Citations

H-Index

Page 1

	Year	Citations
Inhibition of autophagy inhibits the conversion of cardiac fibroblasts to cardiac myofibroblasts Shivika Gupta, Matthew R. Zeglinski, Sunil G. Rattan, Oncotarget	2016	66
An Improved Method of Maintaining Primary Murine Cardiac Fibroblasts in Two-Dimensional Cell Culture Natalie M. Landry, Sunil G. Rattan, Ian Dixon Scientific Reports	2019	64
Scleraxis and fibrosis in the pressure-overloaded heart Raghu S. Nagalingam, Sikta Chattopadhyaya, Danah S. Al‐Hattab, European Heart Journal	2022	46
SKI activates the Hippo pathway via LIMD1 to inhibit cardiac fibroblast activation Natalie M. Landry, Sunil G. Rattan, Krista L. Filomeno, Basic Research in Cardiology	2021	37
Regulation of cardiac fibroblast MMP2 gene expression by scleraxis Raghu S. Nagalingam, Hamza A. Safi, Danah S. Al‐Hattab, Journal of Molecular and Cellular Cardiology Transcriptional RegulationFibrosisCardiomyopathySclerodermaVascular Biology	2018	20
The Functional Role of Zinc Finger E Box-Binding Homeobox 2 (Zeb2) in Promoting Cardiac Fibroblast Activation Fahmida Jahan, Natalie M. Landry, Sunil G. Rattan, International Journal of Molecular Sciences Cardiac MuscleHeart FailureCardiac Progenitor CellsFunctional RoleCardiac Regeneration	2018	14
Ski drives an acute increase in MMP-9 gene expression and release in primary cardiac myofibroblasts Natalie M. Landry, Morvarid Kavosh, Krista L. Filomeno, Physiological Reports	2018	10
Elimination of endogenous high molecular weight FGF2 prevents pressure-overload-induced systolic dysfunction, linked to increased FGFR1 activity and NR1D1 expression Navid Koleini, Barbara E. Nickel, Raghu S. Nagalingam, Cell and Tissue Research	2021	10

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