The effects of secreted aspartyl proteinase inhibitor ritonavir on azoles‐resistant strains of <i>Candida albicans</i> as well as regulatory role of <i>SAP2</i> and <i>ERG11</i>

Abstract

These findings may help to improve our understanding of azole-resistant mechanisms of C. albicans and provide a novel direction for clinical therapeutics of C. albicans infection.