Publication | Closed Access
Targeting KEAP1/Nrf2, AKT, and PPAR-γ signals as a potential protective mechanism of diosmin against gentamicin-induced nephrotoxicity
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Citations
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References
2021
Year
InflammationMolecular PharmacologyPotential Protective MechanismAnti-inflammatoryMedicineGentamicin-induced NephrotoxicityImmunologyCellular PharmacologyHepatotoxicityRenal PathophysiologyPharmacologyDrug DiscoveryOxidative StressPpar-γ Signals
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