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Chlorogenic acid abates male reproductive dysfunction in arsenic-exposed mice via attenuation of testicular oxido-inflammatory stress and apoptotic responses

InfertilityAndrologyArsenic-exposed MiceFertilityTesticular Oxido-inflammatory StressMedicinePhysiologyReproductive HealthGynecologyToxicologyReproductive BiologyPublic HealthEndocrinologyApoptotic ResponsesToxicological MechanismReproductive HormoneOxidative StressReproductive Physiology