Extracellular matrix remodelling is associated with muscle force increase in overloaded mouse <i>plantaris</i> muscle

Abstract

ECM remodelling is associated with an increase in muscle mass and force in overload-modelled dystrophic muscle. Lack of myostatin is not advantageous and inhibition of ECM deposition by halofuginone is disadvantageous for muscle plasticity in response to stimuli that induce dystrophic muscle.