Publication | Closed Access

IFN-γ Activates the TLR4-CCL5 Signaling Through Reducing Arginine Level, Leading to Enhanced Susceptibility of Bovine Mammary Epithelial Cells to Staphylococcus aureus

InflammationMicrobial PathogensCell SignalingPathogenesisImmune RegulationImmunologyStaphylococcus AureusTlr4-ccl5 SignalingInnate Immune SystemMammary GlandInnate ImmunityInfection ControlReducing Arginine LevelImmune SystemMedicineClinical Microbiology