Publication | Open Access

Chemical hypoxia induces pro-inflammatory signals in fat-laden hepatocytes and contributes to cellular crosstalk with Kupffer cells through extracellular vesicles

Lipid PeroxidationImmunologyFatty Liver DiseaseCellular PhysiologyOxidative StressInflammationHepatotoxicityCell SignalingHealth SciencesLiver PhysiologyChronic InflammationChemical HypoxiaCell BiologyExtracellular VesiclesHepatologyPhysiologyMetabolic RegulationKupffer CellsMetabolismMedicine