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Loss of tissue-nonspecific alkaline phosphatase (TNAP) enzyme activity in cerebral microvessels is coupled to persistent neuroinflammation and behavioral deficits in late sepsis

Cerebral Vascular RegulationNeurophysiologyMedicinePhysiologyBrain-immune InteractionSocial SciencesBrain InjuryNeurologyNeuroprotectionNeuroscienceNervous SystemCerebral Blood FlowNeuroimmunologyCerebral MicrovesselsLate SepsisBehavioral DeficitsDisease PhysiologyNeuroinflammation