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PM2.5 promotes plaque vulnerability at different stages of atherosclerosis and the formation of foam cells via TLR4/MyD88/NFκB pathway

InflammationVascular DiseaseCardiovascular DiseaseFoam CellsImmunologyEndothelial DysfunctionChronic InflammationVascular BiologyDifferent StagesCell BiologyMedicineCardiovascular Disease PathogenesisAtherosclerosisPlaque Vulnerability