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Brain permeant and impermeant inhibitors of fatty-acid amide hydrolase suppress the development and maintenance of paclitaxel-induced neuropathic pain without producing tolerance or physical dependence in vivo and synergize with paclitaxel to reduce tumor cell line viability in vitro

Paclitaxel-induced Neuropathic PainNeuro-oncologyHealth SciencesPain MedicineMedicineFatty-acid Amide HydrolaseNeuropathic PainCancer PainNeuropharmacologyMolecular PainPain ManagementPharmacotherapyNeurosciencePain MechanismPharmacologyGliomaBrain PermeantPain Research