Publication | Closed Access

Sestrin2 increases in aortas and plasma from aortic dissection patients and alleviates angiotensin II-induced smooth muscle cell apoptosis via the Nrf2 pathway

InflammationVascular DiseaseNrf2 PathwayCardiovascular DiseaseAortic Dissection PatientsMedicinePhysiologyEndothelial DysfunctionCardiovascular PharmacologyCell DeathVascular BiologyPharmacologyAtherosclerosisAortic DissectionOxidative Stress