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Loss of RIP3 initiates annihilation of high-fat diet initialized nonalcoholic hepatosteatosis: A mechanism involving Toll-like receptor 4 and oxidative stress

InflammationHepatologyBiochemistryLiver PhysiologyLipid PeroxidationAutophagyImmunologyCell DeathNonalcoholic HepatosteatosisHepatotoxicityFatty Liver DiseaseMetabolismMedicineToll-like Receptor 4Lipid SynthesisOxidative Stress