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VEGF165b, a splice variant of VEGF-A, promotes lung tumor progression and escape from anti-angiogenic therapies through a β1 integrin/VEGFR autocrine loop

Lung Tumor ProgressionAngiogenesisMedicineSplice VariantAnti-angiogenic TherapiesPathologyCancer BiologyVascular BiologyOncologyCell BiologyLung CancerTumor BiologyCancer Growth