Prion protein stabilizes amyloid-β (Aβ) oligomers and enhances Aβ neurotoxicity in a Drosophila model of Alzheimer's disease

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Overdependence on degraded gist memory in Alzheimer's disease.

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Neuropsychology, Neurolinguistics, Cognition +18

Naturally secreted oligomers of amyloid β protein potently inhibit hippocampal long-term potentiation in vivo

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Diffusible, nonfibrillar ligands derived from Aβ _1–42 are potent central nervous system neurotoxins

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RETRACTED ARTICLE: A specific amyloid-β protein assembly in the brain impairs memory

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