Maj-Linda B. Selenica

Author

USF Health Byrd Alzheimer's Institute

Also Known As

Maj-Linda B Selenica, Maj-Linda B. Selenica, Maj-Linda Selenica

Publications

11.2K

Citations

H-Index

Concepts

All Affiliations

USF Health Byrd Alzheimer's Institute University of South Florida Johns Hopkins University Johns Hopkins Medicine

About

Maj-Linda B. Selenica is an author at USF Health Byrd Alzheimer's Institute specializing in medicine, alzheimer's disease, and neuroscience.

Top Concepts

#	Concept	H-Index	Publications	Citations
1	Medicine	5	5	593
2	Immunology	2	2	297
3	Health Sciences	1	1	119
4	Social Sciences	1	1	71
5	Allergy	1	1	106

Maj-Linda B. Selenica

Publications

11.2K

Citations

H-Index

Page 1

	Year	Citations
Amyloid Oligomers Exacerbate Tau Pathology in a Mouse Model of Tauopathy Maj-Linda B. Selenica, Milene L. Brownlow, Jeffy P. Jimenez, Neurodegenerative Diseases	2012	10.4K
LPS- induced inflammation exacerbates phospho-tau pathology in rTg4510 mice Daniel C. Lee, Justin Rizer, Maj-Linda B. Selenica, Journal of Neuroinflammation ImmunologyCell DeathVehicle InjectionsAmyloid Precursor ProteinNeuroinflammation	2010	285
Histone deacetylase 6 inhibition improves memory and reduces total tau levels in a mouse model of tau deposition Maj-Linda B. Selenica, Leif Benner, Steven B. Housley, Alzheimer s Research & Therapy	2014	130
Aging enhances classical activation but mitigates alternative activation in the central nervous system Daniel C. Lee, Claudia R. Ruiz, Lori Lebson, Neurobiology of Aging Alternative ActivationEnhances Classical ActivationLifespan ExtensionAgingAnti-aging	2013	119
Fractalkine overexpression suppresses tau pathology in a mouse model of tauopathy Kevin Nash, Daniel C. Lee, Jerry B. Hunt, Neurobiology of Aging AllergyTau PathologyPathologyDegenerative PathologyMedicine	2013	106
Diverse activation of microglia by chemokine (C-C motif) ligand 2 overexpression in brain Maj-Linda B. Selenica, Jennifer A. Alvarez, Kevin Nash, Journal of Neuroinflammation	2013	92
Partial rescue of memory deficits induced by calorie restriction in a mouse model of tau deposition Milene L. Brownlow, Aurélie Joly‐Amado, Sana Azam, Behavioural Brain Research Memory DeficitsEnergy HomeostasisAlzheimer's DiseaseMemory SystemMedicine	2014	71
Chronological Age Impacts Immunotherapy and Monocyte Uptake Independent of Amyloid Load Qingyou Li, Lori Lebson, Daniel C. Lee, Journal of Neuroimmune Pharmacology Alzheimer's DiseaseAgingLongevityDementiaImmunology	2011	12

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