Publication | Closed Access

Induction of inactive TGF-β1 monomer formation by hydrogen sulfide contributes to its suppressive effects on Ang II- and TGF-β1-induced EMT in renal tubular epithelial cells

UrologyRenal FunctionHydrogen SulfideRenal PathologyRenal InflammationTgf-β1-induced EmtRenal PathophysiologyKidney Tubule RemodelingMedicineCell BiologyCell SignalingNephrologyKidney ResearchAng Ii-