<i>N</i>‐Acetylcysteine Reduces ROS‐Mediated Oxidative DNA Damage and PI3K/Akt Pathway Activation Induced by <i>Helicobacter pylori</i> Infection

Abstract

An increased ROS level is a critical mechanism in <i>H. pylori</i> pathogenesis, and NAC may be beneficial for the treatment of <i>H. pylori</i>-related gastric diseases linked to oxidative DNA damage.