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CaMKII-mediated phosphorylation of RyR2 plays a crucial role in aberrant Ca2+ release as an arrhythmogenic substrate in cardiac troponin T-related familial hypertrophic cardiomyopathy

Cardiac MuscleCardiomyopathyHeart FailureMolecular PhysiologySignal TransductionMedicinePhysiologyMolecular BiologyCamkii-mediated PhosphorylationCardiologyAberrant Ca2+ ReleaseCardiovascular FunctionArrhythmogenic SubstrateCell SignalingCellular Physiology