Publication | Open Access

Microglial activation and the nitric oxide/cGMP/PKG pathway underlie enhanced neuronal vulnerability to mitochondrial dysfunction in experimental multiple sclerosis

InflammationMicroglial ActivationMitochondrial DysfunctionMedicineImmunologyCell DeathBrain-immune InteractionNeuroprotectionNeuroscienceNeurologyNeurodegenerationMultiple SclerosisExperimental Multiple SclerosisNeuroimmunologyNeuroinflammation