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Methylglyoxal-derived advanced glycation end products contribute to negative cardiac remodeling and dysfunction post-myocardial infarction

CardiomyopathyHeart FailureNegative Cardiac RemodelingDysfunction Post-myocardial InfarctionBiochemistryCardiovascular DiseaseMedicineGlycobiologyCardiac RegenerationCardiologyAcute Myocardial InfarctionGlycation End ProductsPharmacologyAtherosclerosisBiomolecular EngineeringOxidative StressMyocardial Infarction