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VDAC1 deacetylation is involved in the protective effects of resveratrol against mitochondria-mediated apoptosis in cardiomyocytes subjected to anoxia/reoxygenation injury

Reductive StressBiochemistryRedox RegulatorMedicineLipid PeroxidationCell DeathProtective EffectsVdac1 DeacetylationReactive Oxygen SpeciePharmacologyCell BiologyRedox BiologyMitochondria-mediated ApoptosisOxidative Stress