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Rifampicin-induced injury in HepG2 cells is alleviated by TUDCA via increasing bile acid transporters expression and enhancing the Nrf2-mediated adaptive response

InflammationAnti-inflammatorySignal TransductionMedicineLiver PhysiologyHepg2 CellsImmunologyLipid PeroxidationCell DeathNrf2-mediated Adaptive ResponseHepatotoxicityPharmacologyCell BiologyDrug-induced Liver InjuryRifampicin-induced InjuryOxidative Stress