<scp>KLF</scp>4 Knockdown Attenuates <scp>TBI</scp>‐Induced Neuronal Damage through p53 and <scp>JAK</scp>‐<scp>STAT</scp>3 Signaling

Abstract

These findings provide evidence that KLF4 plays an important role in the pathophysiology of TBI. Blocking KLF4 may be a potential therapeutic strategy for the treatment of TBI, either alone or in combination with agents that target complementary mechanisms.