Licarin B from Myristica fragrans improves insulin sensitivity via PPARγ and activation of GLUT4 in the IRS-1/PI3K/AKT pathway in 3T3-L1 adipocytes

Abstract

Licarin B, a major bioactive compound isolated from<italic>Myristica fragrans</italic>mace improves glucose uptake<italic>via</italic>PPARγ and GLUT4 translocation in IRS-1/PI3K/AKT pathway in adipocytes