Publication | Open Access

The ATG16L1 risk allele associated with Crohn's disease results in a Rac1-dependent defect in dendritic cell migration that is corrected by thiopurines

InflammationDendritic Cell MigrationAutoimmune DiseaseMucosal ImmunologyImmunologyImmune RegulationAutoimmunityRac1-dependent DefectMedicineCell BiologyCell SignalingInborn Error Of ImmunityImmune DysregulationAtg16l1 Risk