Publication | Open Access
Pulmonary receptor for advanced glycation end-products promotes asthma pathogenesis through IL-33 and accumulation of group 2 innate lymphoid cells
105
Citations
66
References
2015
Year
InflammationAsthmaCytokineGroup 2AllergyInflammatory Lung DiseaseLung InflammationInnate Lymphoid CellsImmunologyImmunologic MechanismPulmonary ReceptorInnate ImmunityImmunopathologyMedicineCell Biology
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