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High levels of endogenous nitric oxide produced after burn injury in rats arrest activated T lymphocytes in the first G1 phase of the cell cycle and then induce their apoptosis

InflammationAcute Lung InjuryMedicinePhysiologyApoptosisImmunologyCell DeathBurn ManagementBurn InjuryCell CycleEndogenous Nitric OxideCell BiologyNitrosative StressOxidative Stress