Publication | Open Access

Endotoxin-induced vascular endothelial cell migration is dependent on TLR4/NF-κB pathway, NAD(P)H oxidase activation, and transient receptor potential melastatin 7 calcium channel activity

ImmunologyCellular PhysiologyOxidative StressInflammationCell SignalingMolecular SignalingEndothelial Cell PathobiologyVascular AdaptationVascular PharmacologyTlr4/nf-κb PathwayVascular BiologyH Oxidase ActivationCell BiologyCalcium Channel ActivitySignal TransductionPhysiologyEndothelial DysfunctionCell MigrationMedicine