Publication | Open Access

Two distinct functional sites of human interleukin 4 are identified by variants impaired in either receptor binding or receptor activation.

InflammationCytokineMolecular ImmunologyImmunogeneticsAllergyAutoimmune DiseaseDistinct Functional SitesImmune Cell DevelopmentImmunologyAutoimmunityCd4 T Cell ResponsesReceptor BindingImmunopathologyMedicineReceptor Activation