Publication | Closed Access
Interleukin-18-induced atherosclerosis involves CD36 and NF-κB crosstalk in Apo E−/− mice
66
Citations
29
References
2014
Year
InflammationCytokineApo E−/− MiceImmune Cell DevelopmentImmunologyImmune RegulationChronic InflammationNf-κb CrosstalkInterleukin-18-induced AtherosclerosisVascular BiologyImmune FunctionCell BiologyImmune SystemMedicineInflammatory DiseaseAtherosclerosisImmune Dysregulation
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