Advanced glycation endproduct-modified superoxide dismutase-1 (SOD1)-positive inclusions are common to familial amyotrophic lateral sclerosis patients with SOD1 gene mutations and transgenic mice expressing human SOD1 with a G85R mutation

Publication | Closed Access

DOI

Citations

References

2000

Year

S. Kato, Seiko Horiuchi, Jian Liu, Don W. Cleveland, Noriyuki Shibata, Kenji Nakashima, Ryoji Nagai, Asao Hirano, Miki Takikawa, Masako Kato,

Acta Neuropathologica

Amyotrophic Lateral SclerosisMolecular NeuroscienceG85r MutationSod1 Gene MutationsDegenerative PathologyNeurologyHuman Sod1NeurodegenerationMedicineCell Biology