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Bone marrow stromal cells attenuate sepsis via prostaglandin E2–dependent reprogramming of host macrophages to increase their interleukin-10 production

InflammationCytokineInflammatory DiseaseInterleukin-10 ProductionImmune Cell DevelopmentImmunologyImmune RegulationPathologyChronic InflammationImmune SurveillanceHost MacrophagesImmunologic MechanismPhagocyteImmune SystemMedicineCell BiologyProstaglandin E2–dependent Reprogramming