Publication | Open Access

Gap-filling and bypass at the replication fork are both active mechanisms for tolerance of low-dose ultraviolet-induced DNA damage in the human genome

ChromatinGenome InstabilityDna DamageNatural SciencesGeneticsGenomic MechanismMolecular BiologyDna ReplicationReplication ForkNuclear OrganizationMolecular GeneticsActive MechanismsChromosomal RearrangementHuman GenomeMedicineCell Biology