Publication | Open Access

Double-Stranded RNA-Exposed Human Keratinocytes Promote Th1 Responses by Inducing a Type-1 Polarized Phenotype in Dendritic Cells: Role of Keratinocyte-Derived Tumor Necrosis Factor α, Type I Interferons, and Interleukin-18

Innate Immune SystemImmunologyImmune RegulationImmunologic MechanismInnate ImmunityDermatologyImmune SystemImmune DysregulationInflammationType-1 Polarized PhenotypeCell SignalingT Cell ImmunityImmune FunctionDendritic CellsCytokineImmune Cell DevelopmentDendritic Cell BiologyMedicineViral Immunity