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Successful reversal of propionic acidaemia associated cardiomyopathy: Evidence for low myocardial coenzyme Q10 status and secondary mitochondrial dysfunction as an underlying pathophysiological mechanism

Cardiac MuscleCardiomyopathyHeart FailureCardiovascular DiseaseMitochondrial FunctionBiochemistrySuccessful ReversalPhysiologyPropionic AcidaemiaSecondary Mitochondrial DysfunctionMetabolismMedicineCardiologyOxidative Stress