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Early neuronal dysfunction by amyloid β oligomers depends on activation of NR2B-containing NMDA receptors

Amyloid β OligomersSynaptic PlasticityNeurodegenerative DiseasesMolecular NeuroscienceSynaptic TransmissionProtein MisfoldingEarly Neuronal DysfunctionNeurologyNeuroscienceMolecular NeurobiologyNr2b-containing Nmda ReceptorsMedicineSocial Sciences