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Influence of cytosolic and mitochondrial Ca2+, ATP, mitochondrial membrane potential, and calpain activity on the mechanism of neuron death induced by 3-nitropropionic acid

NeurophysiologyMedicineApoptosisPhysiologyMitochondrial Ca2+Cell DeathNeuroprotectionNeuroscienceCalpain ActivityNervous SystemPharmacologyCellular PhysiologySocial SciencesOxidative StressMitochondrial Membrane Potential