Absence of an Orphan Mitochondrial Protein, C19orf12, Causes a Distinct Clinical Subtype of Neurodegeneration with Brain Iron Accumulation - Concepedia

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Absence of an Orphan Mitochondrial Protein, C19orf12, Causes a Distinct Clinical Subtype of Neurodegeneration with Brain Iron Accumulation

DOI Full Paper Access

262

Citations

15

References

2011

Year

Monika Hartig, Arcangela Iuso, Tobias B. Haack, Tomasz Kmieć, Elżbieta Jurkiewicz, Katharina Heim, Sigrun Roeber, Victoria Tarabin, Sabrina Dusi, Małgorzata Krajewska‐Walasek,

The American Journal of Human Genetics

Neurodegenerative DiseasesAlzheimer's DiseaseMitochondrial FunctionBiochemistryIron MetabolismDegenerative PathologyBrain Iron AccumulationNeurochemical BiomarkersNeuroprotectionNeuroscienceNeurologyNeurodegenerationDistinct Clinical SubtypeNeuropathologyMedicineOrphan Mitochondrial ProteinOxidative Stress

References

	Year	Citations

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