Publication | Open Access
Opposite effects of the p52shc/p46shc and p66shc splicing isoforms on the EGF receptor-MAP kinase-fos signalling pathway
393
Citations
51
References
1997
Year
Opposite EffectsMolecular PhysiologySignal TransductionSignaling PathwayCell RegulationP66shc Splicing IsoformsNatural SciencesReceptor Tyrosine KinaseMolecular BiologyCellular BiochemistryMedicineCell BiologyCell SignalingProtein Phosphorylation
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