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Gallic acid attenuates high-fat diet fed-streptozotocin-induced insulin resistance via partial agonism of PPARγ in experimental type 2 diabetic rats and enhances glucose uptake through translocation and activation of GLUT4 in PI3K/p-Akt signaling pathway

Metabolic SyndromeInsulin SignalingBiochemistryMedicinePartial AgonismPhysiologyDiabetesInsulin ManagementGallic AcidMetabolic RegulationMetabolismGlycemic ResponseDiabetes MellitusMetabolic SignalingPharmacologyExperimental Type 2Metabolic HealthHealth Sciences