Oxidative stress and increased expression of growth factors in lesions of failed hemodialysis access

Advanced oxidation protein products as a novel marker of oxidative stress in uremia

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Enhanced cellular oxidant stress by the interaction of advanced glycation end products with their receptors/binding proteins

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Vascular access for hemodialysis

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Redox-mediated activation of latent transforming growth factor-beta 1.

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Human neutrophils employ the myeloperoxidase-hydrogen peroxide-chloride system to convert hydroxy-amino acids into glycolaldehyde, 2-hydroxypropanal, and acrolein. A mechanism for the generation of highly reactive alpha-hydroxy and alpha,beta-unsaturated aldehydes by phagocytes at sites of inflammation.

M M Anderson, Stanley L. Hazen, Fong‐Fu Hsu et al. · Journal of Clinical Investigation · 1997 · 389 citations · Full text

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