Corticostriatal dysfunction underlies diminished striatal ascorbate release in the R6/2 mouse model of Huntington's disease

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Characterization of Progressive Motor Deficits in Mice Transgenic for the Human Huntington’s Disease Mutation

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A family of mammalian Na+-dependent L-ascorbic acid transporters

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A theory of the functional organization of the neostriatum and the neostriatal control of voluntary movement

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Altered brain neurotransmitter receptors in transgenic mice expressing a portion of an abnormal human Huntington disease gene

J. Jang‐Ho, Christoph M. Kosinski, Julie A. Kerner et al. · Proceedings of the National Academy of Sciences · 1998 · 502 citations · Full text