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Impaired relaxation to acetylcholine in 2K-1C hypertensive rat aortas involves changes in membrane hyperpolarization instead of an abnormal contribution of endothelial factors

HypertensionMolecular PhysiologyCardiovascular DiseaseMedicineVascular AdaptationPhysiologyEndothelial DysfunctionVascular BiologyElectrophysiologyCardiovascular PhysiologyMembrane HyperpolarizationCardiovascular FunctionPharmacologyImpaired RelaxationAtherosclerosisEndothelial Factors