Gláucia E. Callera - Concepedia

Author

Gláucia E. Callera

Also Known As

G E Callera, G. Callera, G. E. Callera, G.E. Callera, Glaucia Callera, Glaucia E Callera, Glaucia E. Callera, Glaucia Elena Callera, Glaucia E. Callera, Gláucia E Callera

Publications

4.2K

Citations

H-Index

Concepts

All Affiliations

University of Ottawa Universidade de São Paulo Ottawa Hospital University of Glasgow Universidade de Ribeirão Preto

About

Gláucia E. Callera is an author at University of Ottawa specializing in physiology, medicine, and vascular biology.

Top Concepts

#	Concept	H-Index	Publications	Citations
1	Medicine	20	22	1.9K
2	Cellular Physiology	8	8	755
3	Cell Biology	7	7	817
4	Health Sciences	5	5	619
5	Biochemistry	5	5	542

Gláucia E. Callera

Publications

3.9K

Citations

H-Index

Page 1

	Year	Citations
Critical role of Nox4-based NADPH oxidase in glucose-induced oxidative stress in the kidney: implications in type 2 diabetic nephropathy Mona Sedeek, Gláucia E. Callera, Augusto C. Montezano, American Journal of Physiology-Renal Physiology	2010	384
Adipocytes Produce Aldosterone Through Calcineurin-Dependent Signaling Pathways Ana M. Briones, Aurélie Nguyen Dinh Cat, Gláucia E. Callera, Hypertension Insulin SignalingObesityInflammationMetabolic SyndromeMolecular Pharmacology	2012	353
Aldosterone Activates Vascular p38MAP Kinase and NADPH Oxidase Via c-Src Gláucia E. Callera, Rhian M. Touyz, Rita C. Tostes, Hypertension Aldosterone EffectsAldo-keto ReductaseSelective Src InhibitorP38map KinaseRedox Biology	2005	245
Angiotensin(1-7) Blunts Hypertensive Cardiac Remodeling by a Direct Effect on the Heart Chantal Mercure, Álvaro Yogi, Gláucia E. Callera, Circulation Research HypertensionHeart FailureCardiovascular DiseaseAtherosclerosisAce2 Inactivation	2008	225
Nicotinamide Adenine Dinucleotide Phosphate Reduced Oxidase 5 (Nox5) Regulation by Angiotensin II and Endothelin-1 Is Mediated via Calcium/Calmodulin-Dependent, Rac-1-Independent Pathways in Human Endothelial Cells Augusto C. Montezano, Dylan Burger, Tamara M. Paravicini, Circulation Research	2010	185
Aldosterone and Angiotensin II Synergistically Stimulate Migration in Vascular Smooth Muscle Cells Through c-Src-Regulated Redox-Sensitive RhoA Pathways Augusto C. Montezano, Gláucia E. Callera, Álvaro Yogi, Arteriosclerosis Thrombosis and Vascular Biology	2008	162
ET <sub>A</sub> Receptor Blockade Decreases Vascular Superoxide Generation in DOCA-Salt Hypertension Gláucia E. Callera, Rhian M. Touyz, Simone A. Teixeira, Hypertension	2003	139
Angiotensin II-Dependent Chronic Hypertension and Cardiac Hypertrophy Are Unaffected by gp91phox-Containing NADPH Oxidase Rhian M. Touyz, Chantel Mercure, Ying He, Hypertension HypertensionCardiovascular PharmacologyAng IiCardiovascular FunctionReactive Oxygen Species	2005	131
Endothelin-1-induced oxidative stress in DOCA-salt hypertension involves NADPH-oxidase-independent mechanisms Gláucia E. Callera, Rita C. Tostes, Álvaro Yogi, Clinical Science	2006	119
Testosterone Induces Vascular Smooth Muscle Cell Migration by NADPH Oxidase and c-Src–Dependent Pathways Andréia Z. Chignalia, Elke Zuleika Schuldt, Lívia L. Camargo, Hypertension HypertensionNadph OxidaseRedox BiologyCellular PhysiologyOxidative Stress	2012	115

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