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Nicotine restores endothelial dysfunction caused by excess sFlt1 and sEng in an in vitro model of preeclamptic vascular endothelium: a possible therapeutic role of nicotinic acetylcholine receptor (nAChR) agonists for preeclampsia

HypertensionMedicineNicotinePhysiologyEndothelial DysfunctionPreeclamptic Vascular EndotheliumNeuropeptide ReceptorVascular BiologyExcess Sflt1PharmacologyPreeclampsiaCerebral Vascular Regulation