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The loss of glucose-regulated protein 78 (GRP78) during normal aging or from siRNA knockdown augments human alpha-synuclein (α-syn) toxicity to rat nigral neurons

Neurodegenerative DiseasesAlzheimer's DiseaseAgingNeurobiology Of DiseaseNigral NeuronsSirna Knockdown AugmentsProtein DegradationDegenerative PathologyNeural AgingNeuroprotectionNeurologyNeuroscienceNeurodegenerationMedicineCell BiologySocial SciencesGlucose-regulated Protein 78